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By Fangli Zheng

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TDAG8 is a proton-sensing and psychosine-sensitive G-protein-coupled receptor. J Biol Chem 279: 45626–45633. [30] Xi Q. et al. a new mechanism improving the survival and metastasis of tumor cell by proton-sensing receptor TDAG8 in the acidic condition (unpublished). G. et al. 2008. Acid-sensing ion channels (ASICs) as pharmacological targets for neurodegenerative diseases. Current Opinion in Pharma-cology 8: 25–32. [32] Yoneda, T. et al. 2011. Involvement of acidic microenvironment in the pathophysiology of cancer-associated bone pain.

Journal of China Coal Society 21(1): 63–67. [10] Nie, BS. & He, XQ et al. 2004. Reseach on the mechanisms of the influence of electromagnetic field on coalbed mathane adsorpion. Natural Gas Industry 24(10): 32–34. [11] Jiang, YD. & Yang, XY et al. 2010. The infiltration equation of coal bed under the cooperation of stress field, temperature field and sound field. Journal of China Coal Society 35(3): 434–438. [12] Jiang, YD. & Xian, XF et al. 2008. Experimental and mechanical on the features of ultrasonic vibration stimulating the desorption ofmethane in coal.

This result proved that TDAG8 can promote the formation of tumor alone in the absence of other proton sensory receptors. Hang et al. (2012) observed the effect of siRNA (small interfering RNA) intrathecal injection of TDAG8 injected on mechanical hyperalgesia in Rat Model of Bone Cancer Pain and spinal cord TDAG8 expression. 01). 01). It was proved that the spinal cord TDAG8 may be involved in the formation and development of bone cancer pain in rats and the intrathecal injection of TDAG8’s interferential siRNA can relief pain.

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