Download Evidence-based Gastroenterology and Hepatology, Second PDF

Conventional textbooks during this box have emphasised the elemental sciences of pathology, biochemistry and body structure. Evidence-based Gastroenterology and Hepatology covers all of the significant illnesses of the gastrointestinal tract and liver, using scientific epidemiology to provide the most powerful and most modern facts for interventions.

This moment version is edited and written by means of top gastroenterologists from world wide, every one bankruptcy summarizes the proof in order that larger expert judgements may be made approximately which remedies to supply to patients.

It presents practicing Gastroenterologists and Surgeons with transparent information about the prognosis and therapy of pancreatic illnesses, giving transparent proof and experience-based fabric that's instantly proper to medical practice.

Also encompasses a checklist of steered analyzing on the finish of every chapter.

Take a glance at modern info at www.evidbasedgastro.com

Content:
Chapter 1 creation (pages 1–11): John WD McDonald, Brian G Feagan and Andrew okay Burroughs
Chapter 2 Gastroesophageal Reflux disorder (pages 13–54): Naoki Chiba
Chapter three Barrett's Esophagus (pages 55–68): Carlo A Fallone, Marc Bradette and Naoki Chiba
Chapter four Esophageal Motility problems: Achalasia and Spastic Motor issues (pages 69–81): Marcelo F Vela and Joel E Richter
Chapter five Ulcer sickness and Helicobacter Pylori (pages 83–116): Naoki Chiba
Chapter 6 Non?Steroidal Anti?Inflammatory Drug?Induced Gastroduodenal Toxicity (pages 117–138): Alaa Rostom, Andreas Maetzel, Peter Tugwell and George Wells
Chapter 7 Non?Variceal Gastrointestinal Hemorrhage (pages 139–159): Nicholas Church and Kelvin Palmer
Chapter eight useful Dyspepsia (pages 161–168): Sander JO Veldhuyzen van Zanten
Chapter nine Celiac ailment (pages 169–178): James Gregor and Diamond Sherin Alidina
Chapter 10 Crohn's sickness (pages 179–195): Brian G Feagan and John WD McDonald
Chapter eleven Ulcerative Colitis (pages 197–210): Derek P Jewell and Lloyd R Sutherland
Chapter 12 Pouchitis After Restorative Proctocolectomy (pages 211–219): William J Sandborn
Chapter thirteen Microscopic and Collagenous Colitis (pages 221–229): Robert Lofberg
Chapter 14 Metabolic Bone affliction in Gastrointestinal issues (pages 231–246): Ann Cranney, Catherine Dube, Alaa Rostom, Peter Tugwell and George Wells
Chapter 15 Colorectal melanoma in Ulcerative Colitis: Surveillance (pages 247–253): Bret A Lashner and Alastair JM Watson
Chapter sixteen Colorectal melanoma: inhabitants Screening and Surveillance (pages 255–263): Bernard Levin
Chapter 17 Irritable Bowel Syndrome (pages 265–283): Albena Halpert and Douglas A Drossman
Chapter 18 Clostridium Difficile illness (pages 285–301): Lynne V McFarland and Christina M Surawicz
Chapter 19 Ogilvie's Syndrome (pages 303–309): Michael D Saunders and Michael B Kimmey
Chapter 20 Gallstone affliction (pages 311–320): Calvin HL legislations, Dana McKay and Ved R Tandan
Chapter 21 Acute Pancreatitis (pages 321–339): Jonathon Springer and Hillary Steinhart
Chapter 22 weight problems (pages 341–357): Jarol Knowles
Chapter 23 Hepatitis C (pages 359–366): Patrick Marcellin
Chapter 24 Hepatitis B (pages 367–381): Piero Almasio, Calogero Camma, Vito Di Marco and Antonio Craxi
Chapter 25 Alcoholic Liver affliction (pages 383–391): Philippe Mathurin and Thierry Poynard
Chapter 26 Non?Alcoholic Fatty Liver ailment (pages 393–403): Chris P Day
Chapter 27 Hemochromatosis and Wilson illness (pages 405–413): Gary Jeffrey and Paul C Adams
Chapter 28 fundamental Biliary Cirrhosis (pages 415–426): Jenny Heathcote
Chapter 29 Autoimmune Hepatitis (pages 427–434): Michael Peter Manns and Andreas Schuler
Chapter 30 fundamental Sclerosing Cholangitis (pages 435–451): Roger Chapman and Sue Cullen
Chapter 31 Portal Hypertensive Bleeding (pages 453–485): John Goulis and Andrew ok Burroughs
Chapter 32 Ascites, Hepatorenal Syndrome, and Spontaneous Bacterial Peritonitis (pages 487–503): Pere Gines, Vicente Arroyo and Juan Rodes
Chapter 33 Hepatic Encephalopathy (pages 505–515): Peter Ferenci and Christian Muller
Chapter 34 Hepatocellular Carcinoma (pages 517–525): Massimo Colombo
Chapter 35 Fulminant Hepatic Failure (pages 527–543): Nick Murphy and Julia Wendon
Chapter 36 Liver Transplantation: Prevention and therapy of Rejection (pages 545–571): Laura Cecilioni, Lucy Dagher and Andrew Burroughs
Chapter 37 Liver Transplantation: Prevention and therapy of an infection (pages 573–586): Nancy Rolando and Jim J Wade
Chapter 38 administration of Hepatitis B and C After Liver Transplantation (pages 587–601): George V Papatheodoridis and Rosangela Teixeira

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Additional info for Evidence-based Gastroenterology and Hepatology, Second Edition

Sample text

134 determined that heartburn showed the best discrimination for patients with esophagitis. 135–140 Using the questionnaire developed by Johnsson,135 a positive response to all four questions is required to achieve a high positive predictive value, thus limiting its usefulness. The description of symptoms as opposed to using the term heartburn, may be a factor which improves the predictive value of this questionnaire. 48 The combination of the presence of grade 2 or 3 symptoms on the standardized questionnaire and endoscopic esophagitis, predicted increased acid exposure on 24-hour intraesophageal pH monitoring with a specificity of 97% and a positive predictive value of 98%.

37 Thus, the focus of treatment has been on acid suppression. 23 Evidence-based Gastroenterology Acid secretion can be controlled by various drug classes. Antimuscarinic agents are weak inhibitors of the parietal cell M3 cholinergic receptors and clinical use is limited by anticholinergic side effects. 159 PPIs provide the most potent acid inhibition through covalent binding to the H+, K+-ATPase (acid or proton pump) located in the secretory canaliculus of the parietal cell. Inhibiting the proton pump, which is the final common pathway, blocks acid secretion to all known stimuli.

1 Acid exposure of the distal part of the esophagus during eight 3-hour periods expressed as median % time spent with pH < 4 in 190 patients with different degrees of heartburn and acid regurgitation and 50 asymptomatic endoscopically normal subjects. Reproduced with permission from Joelsson B et al. 131 questionnaire has a maximum score of 18. In the endoscopic comparison, using a threshold of 4, the questionnaire had 70% sensitivity but only 46% specificity for diagnosis of esophagitis. When used in dyspeptic patients, the questionnaire had a sensitivity of 92% but a specificity of only 19% for diagnosis of GERD when compared with abnormal 24-hour intraesophageal pH monitoring.

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