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Inside the axon, a significant percentage of negative charges are bound to large protein molecules that cannot pass through the membrane because of their sizes. Not specifically pertinent to the short-term solution to the HH model is the fact that nerve membrane (on soma, axon, and dendrites), and those of nearly all other types of cells, contains molecular “pumps” driven from the energy in ATP molecules which can eject Na+ from the interior of the cell against the potential energy barrier, ENa, often as an exchange operation with K+ being pumped in.

Electrical synapses allow the depolarization or hyperpolarization of the presynaptic neuron to be directly coupled to the postsynaptic neuron without delays. 5 nm. Penetrating each membrane are curious protein structures called connexons. There are hundreds if not thousands of connexons in each gap junction. Each connexon is made up from six subunits called connexins, spaced around a common center (like the sections of a orange). 5 nm in length, and is directly opposite a corresponding connexin in the opposite membrane.

Choline into the bouton where the enzyme cholineacetyltransferase synthesizes ACh from acetyl coenzyme A and the free choline. This process is more complex that it sounds, because the ACh must be put into the vesicles in just the right amount. When the nicotinic ACh receptor proteins on the SSM each bind with two ACh molecules, their conductance to [Na+] and [K+] increases transiently, allowing sodium ions to flow in and some potassium ions to flow out. The net result of about 200,000 receptor proteins being activated by ACh is the generation of a small transient depolarization of the SSM (an excitatory post synaptic potential or epsp).

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